In addition to the upregulated adipose markers in ILC [17], the inhibition of FABP-4 [28] and CPT-1 [29,30], which were found to be highly expressed in ILC and pleomorphic-type ILC, respectively, have been reported to block tumor growth, indicating that lipid metabolism could be targeted in the context of ILC treatment. The gene discussed is FABP4; the disease is neoplasm.