In addition to the upregulated adipose markers in ILC [17], the inhibition of FABP-4 [28] and CPT-1 [29,30], which were found to be highly expressed in ILC and pleomorphic-type ILC, respectively, have been reported to block tumor growth, indicating that lipid metabolism could be targeted in the context of ILC treatment. This evidence concerns the gene CCL27 and neoplasm.