SLC16A1 and medulloblastoma: Pullen et al. (2011) have also demonstrated in vitro that miR-29b and miR-29c repress Slc16a1. Although, this gene is regulated by miR-124 in medulloblastomas (Li et al., 2009), miR-124 is not expressed at significant levels in mouse islets, at least in young adults, and is thus unlikely to impact Slc16a1 expression in β-cells at this developmental stage (Pullen et al., 2011).