CD14 and rheumatoid arthritis: We also demonstrate that increased mir-155 expression in healthy CD14+ cells renders them more resistant to death and enhances their cytokine/chemokine production These findings suggest that the increased expression of mir-155 in PBM and SFM of patients with RA may contribute to inflammation through two means: first, by increasing monocyte/macrophage survival, possibly by directly repressing expression of pro-apoptotic genes; and second, by increasing levels of a wide range of pro-inflammatory cytokines and chemokines.