ARHGAP5 and nasopharyngeal carcinoma: For example, by down-regulating Bcl-2, miR-744 enhanced apoptosis and inhibited cervical cancer cell proliferation; in hepatocellular carcinoma miR-744 exerted tumor suppressor function by targeting c-Myc [15]; miR-744 was reported to promote the progression of nasopharyngeal carcinoma via directly interacting with the promoter region of ARHGAP5 [20].