Pancreatic inflammation results in destruction of pancreatic islet with loss of β-cells (insulin), α-cells (glucagon), δ-cells (somatostatin) and PP-cells (pancreatic-polypeptide) [2] that can lead to the development of brittle diabetes, which is characterized by extreme blood glucose levels fluctuations, causing hyperglycemia or hypoglycemia [3]. This evidence concerns the gene INS and Hypoglycemia.