NFKB1 and thyroid gland carcinoma: However, when treated withBMS-345541 and 131I, only the NF-κB pathway was suppressed.131I suppressed cell proliferation, induced cell apoptosis, and promotedcell cycle arrest of thyroid cancer cells by up-regulating B-cell translocation gene2-mediated activation of JNK/NF-κB pathways.