Moreover, dendritic cells, MDSCs and tumour‐associated macrophages are all, like CAFs, sources for stromal TGF‐beta (Gabrilovich and Nagaraj, 2009), indicating a mechanism in which TGF‐beta reprogrammes a variety of stromal cells, amplifying its own signal, and impeding anticancer immune responses in a concerted way. Here, TGFB1 is linked to neoplasm.