The significant accumulation of H2O2 in leaves of the SlERF.A1-, SlERF.A3-, SlERF.B4-, and SlERF.C3-silenced plants after infection of B. cinerea (Figure 7) may suggest that the function of SlERF.A1, SlERF.A3, SlERF.B4, and SlERF.C3 in defense response against B. cinerea links to ROS generation. The gene discussed is C3; the disease is infection.