The A549 cells treated with the nuclear factor κB (NF-κB) inhibitor PS-1145, which elevated the Par-4 receptor GRP78 on the cancer cell surface (Burikhanov et al., 2013), were sensitized to apoptosis by the Par-4 secreted in the CM from MEFs in response to CQ treatment (Figure S2A). This evidence concerns the gene NFKB1 and cancer.