Under various stimuli, such as viral infection and apoptosis, FUBP1 could translocate from the nuclei to the cytoplasm, which contributes to mRNA stability and translation, such as GAP-43, p27, and NPM, as well as the replication of viruses, such as the Japanese encephalitis virus and hepatitis C virus [36, 48–52]. The gene discussed is GAP43; the disease is viral infectious disease.