RKIP1 overexpression, for instance, has been shown to suppress EMT in naso‐pharyngeal cancers.25 Members of the Sprouty family are known to repress TGFβ‐induced EMT in the context of cataract development.26 Recent work from the same group has since shown a similar role for Spreds 1–3 and Sef in the same context.27 Furthermore, He et al. 28 recently revealed that hSef plays a role in the negative regulation of EMT in a β‐catenin dependent manner in breast cancer cells. This evidence concerns the gene TGFB1 and pharynx cancer.