2012; Akhuemokhan et al. 2013; Khan and Awan 2014). Zinc was also appreciably detected in the protein isolate. Zinc is known to play a crucial role in antioxidant defense in type 2 diabetic patients where it enhances reduction and neutralization of free radicals and acts as a cofactor of SOD, by modulating glutathione metabolism and metallothionein expression. Its deficiency has been implicated in a number of metabolic abnormalities such as impaired glucose tolerance, decreased pancreatic insulin content as well as insulin degradation (Piero et al. 2012; Cruz et al. 2015). The gene discussed is SOD1; the disease is Impaired glucose tolerance.