Although the number of samples in this study is low, the data are consistent with a heterogeneous distribution of NGFRHigh/Ki‐67Low and NGFRLow/Ki‐67High domains in melanoma tumors, and a drug‐mediated induction of NGFRHigh/Ki‐67Low state that is concomitant with c‐Jun up‐regulation, a situation reminiscent of our observations in cultured cells. Here, JUN is linked to melanoma.