In the present study, the larger N1m source strengths elicited by the tinnitus ear stimulation in the “Silent” condition imply a decrease in parvalbumin-positive interneuron activity, whereas the smaller N1m source strength elicited by the tinnitus ear stimulation in the “Noisy” condition appears to reflect the activity of somatostatin-positive interneurons. The gene discussed is PVALB; the disease is Tinnitus.