Structural defects caused by chromothripsis have been found to recurrently affect ATRX, ODZ3 (odd oz/ten-M homolog 3 (Drosophila)), PTPRD (protein tyrosine phosphatase, receptor type D), and CSMD1 (CUB and sushi multiple domains 1) in high-risk neuroblastoma [35]. This evidence concerns the gene CSMD1 and neuroblastoma.