In a mouse model of diabetic MI, Kishore and colleagues demonstrated that bone marrow-derived progenitor cell therapy contributed to prevention of cardiac fibrosis after MI by releasing hepatocyte growth factor, which inhibited miRNA-155-Ski (Sloan-Kettering Institute proto-oncogene)/SnoN (Ski-related novel gene, non-Alu-containing) mediated profibrosis signaling pathway. The gene discussed is SKIL; the disease is myocardial infarction.