Although FA exposure did not provoke atopic dermatitis in naive rats, overexpression of Th2 cytokines such as IL-4, IL-5 and IL-13, and IL-17A and TSLP in the skin could provide a possible explanation for the potential contribution of FA exposure to the development of atopic dermatitis described in epidemiological studies [12]. The gene discussed is IL4; the disease is atopic eczema.