These data were strongly consistent with the findings obtained in previous studies, which reported a correlation of high SEC62 expression levels with a positive lymph node status in NSCLC patients [23], a stimulation of the migratory potential of HEK293 cells by SEC62 overexpression and inhibition of the migration of prostate cancer, glioblastoma, NSCLC and fibrosarcoma cells by SEC62 silencing [26]. This evidence concerns the gene SEC62 and glioblastoma.