PROS1 and metabolic dysfunction-associated steatotic liver disease: Liver injury from viral hepatitis, alcohol abuse, and non-alcoholic fatty liver diseases/non-alcoholic steatohepatitis (NAFLD/NASH), among others, promotes deposition of extracellular matrix (ECM) protein by myofibroblasts primarily derived from activated hepatic stellate cells (HSCs), the resident pericytes of the liver3.