RAD51D and ovarian serous adenocarcinoma: In addition, recent DNA sequencing studies have revealed the presence of somatic and germline mutations in homologous recombination genes in non-serous ovarian cancers, including some type I lesions; these included somatic mutations in BRCA1, BRCA2, CHEK2, ATM, and germline mutations in BRCA1, BRCA2, RAD51D, CHEK2 and BRIP1, although BRCA1/2 alterations were less common compared with serous ovarian cancers [38,39,40].