We have observed that the T2-high phenotype is a subset of a broader eosinophilic phenotype group in ADEPT and U-BIOPRED studies [27], consistent with results from Choy et al. [28] in mild asthma (evaluating T2 phenotype status by POSTN, SERPINB2, and CLCA1 expression in endobronchial brushings). Here, POSTN is linked to asthma.