Furthermore, this study identified several actionable mutations, including two ETV6–NTRK3 fusions, and FGF6 or FGFR1 alterations, including three FGFR1 fusions and one known intragenic activating FGFR1 mutation, that may significantly impact tumor responses by assisting in the choice of targeted therapies. This evidence concerns the gene NTRK3 and neoplasm.