In addition, Warny et al. [53] described G6PD Herlev variant 592C > A (Arg198Ser) associated with severe enzyme deficiency, prolonged neonatal hyperbilirubinemia, and nonspherocytic hemolytic anemia in a Danish descent male infant who on the second day of life developed jaundice peaking at 67 h that decreased upon application of double-sided phototherapy. The gene discussed is G6PD; the disease is Jaundice.