This can be attributed to the fact that PDAC is a “non‐immunogenic” tumor characterized by low mutational burden (Liu et al, 2011), lack of CD8+ T‐cell infiltration (Ino et al, 2013), and the presence of immunosuppressive myeloid cell populations (Dunn et al, 2002; Stromnes et al, 2015). The gene discussed is CD8A; the disease is neoplasm.