IL37 and myocardial infarction: Inflammatory responses and cytokine expression play an important part in the pathogenesis of post‐MI remodeling.1, 2, 3 Strategies that target inflammation after MI require timely resolution of inflammation without interfering with the healing responses.38 IL‐37 was regarded as a natural suppressor of both innate and adaptive inflammatory and immune responses.12, 13 Furthermore, we previously reported that the plasma IL‐37 concentrations are elevated in patients with acute MI.39 We therefore hypothesized that IL‐37 may participate in ventricular remodeling after MI as a protective mechanism.