Further analyses revealed that altered RNA editing patterns in tumors correlated with ADAR expression, and that non-random, clinically-relevant RNA editing events (frequently located in noncoding RNAs, nonsynonymous sites, intronic regions, and non-Alu elements) correlated with tumor classification and patient survival and with increased cell survival and altered drug sensitivity [2, 3]. The gene discussed is ADAR; the disease is neoplasm.