Given the preferential depletion of Mo-DCs by both anti-GM-CSF [28] and anti-GM-CSFRα administration in the AIP model (Fig. 3b and c), it might have been expected that there would have been a preferential dependence for Mo-DC generation on GM-CSFR signalling in the donor monocytes. Here, CSF2 is linked to autoimmune pancreatitis.