To determine whether this GM-CSF-dependent phenotypic change was actually occurring in recruited monocytes in the inflamed peritoneal cavity we injected MacGreen (Csf1R-EGFP) bone marrow monocytes [28] i.p. at day 2 into C57BL/6 wild-type (WT) recipient mice undergoing AIP, in the presence or absence of CAM-3003 - we previously used this monocyte donor approach to demonstrate the monocyte origin of the AIP CD115+ exudate populations including Mo-DCs [28]. This evidence concerns the gene CSF1R and autoimmune pancreatitis.