MKI67 and endometriosis: Besides, MRC2 significantly induced the differentiation of Treg cells, increased weight of total ectopic lesions, and promoted the expression of Ki-67 in ectopic lesions in vivo, which is consistent with the in vitro results, suggesting that MRC2 is involved in the growth of ectopic lesions and Treg differentiation in endometriosis (Supplementary Figure 4).