Because MUC1 is a heterodimeric protein that is aberrantly expressed in various cancer cells including acute myeloid leukemia (AML) blasts and AML stem cells [48, 49], it is likely that autophagy induced by chemotherapic drugs and small molecular inhibitors in AML enhances the loading of MUC1 onto MHC molecules. The gene discussed is MUC1; the disease is acute myeloid leukemia.