GLO1 and coronary artery disorder: Down regulation of GLO1 in human endothelial cells was associated with a pro-inflammatory and endothelial-activated profile in immortalised HUVEC-derived cells, ECRF-246 and knockdown of GLO1 in human aortic endothelial cells (HAECs) in vitro changed the expression of gene set linked to other coronary artery disease (CAD) associated genes in pathways of lipid metabolism, immune cell regulation, cell stress response, protein metabolism and DNA repair, presuming that GLO1 deficiency is involved in CAD development19.