Type 1 diabetes is a disorder that arises following the autoimmune destruction of insulin-producing pancreatic β cells[1,2] The disease is most often diagnosed in children and adolescents, usually presenting with a classic trio of symptoms (i.e., polydipsia, polyphagia, and polyuria) alongside of overt hyperglycemia, positing the immediate need for exogenous insulin replacement—a medicinal introduction to the disorder whose therapeutic practice lasts a lifetime.[3]. This evidence concerns the gene INS and type 1 diabetes mellitus.