Interestingly, gene expression of FA-transport proteins, Cd36 and Fatp1 was increased in Cpt1bm−/− muscle, suggesting that FA overload on TLRs and FA uptake in Cpt1bm−/− muscle are increased even in CHD condition compared to control mice (Fig. 2b). The gene discussed is SLC27A1; the disease is coronary artery disorder.