MAOA and juvenile absence epilepsy: In PWE with idiopathic generalized epilepsies, like childhood absence epilepsy, JAE, and juvenile myoclonic epilepsy (Blümcke et al.), the functional polymorphism located in the promoter of MAOA gene (MAOA-uVNTR) was evaluated to test whether allelic variation has a role in the etiology of IGE (Haug et al., 2000).