Although, it was expected that the higher activity promoter alleles (3a and 4 copy alleles) would be associated with susceptibility to epilepsy, the frequencies of the high and low (3 copy allele) activity allele groups were similar between PWE and controls, and these results did not confirm any association between the MAOA-uVNTR polymorphism and different IGE subtypes (Haug et al., 2000). Here, MAOA is linked to idiopathic generalized epilepsy.