NFKB1 and acute respiratory distress syndrome: The potential beneficial action of aspirin in ARDS and sepsis has been attributed to its ability to (i) inhibition of COX; (ii) inhibition of nuclear factor kappa B (NFκB); (iii) increase in the production of endothelial nitric oxide (eNO) and inhibition of inducible NO generation; (iv) enhanced production of lipoxin A4 (LXA4) and (v) decrease in the generation of platelet aggregator thromboxane A2 (TXA2) and enhance prostacyclin (PGI2) production [5, 14–20].