LNCARSR and neoplasm: Moreover, RCC cells derived from the shlncARSR-xenografts showed impaired ability to form secondary tumours by serial passage compared to control xenografts (tumour incidence: shGFP, 4/4; shlncARSR-1, 0/4; shlncARSR-2, 0/4) (Supplementary Fig. 2d), indicating that interference of lncARSR impaired the tumour formation ability of renal T-ICs.