In agreement with the above observations, it was found that there was infection-induced secretion of CXCL1 and CXCL2 into the culture supernatants (Fig. 4c,d), which was compromised in the presence of MST1/2 KD constructs (Fig. 4e,f) or Stk4/Stk3 siRNA (Fig. 4g,h). The gene discussed is STK3; the disease is infection.