CPT1, which was up-regulated in the group of animals with high OA content and CPT1B, which was down-regulated in the group of animals with high OA content, are important for the beta-oxidation of long chain fatty acids, which are involved in the long chain fatty acids transport to the mitochondrial matrix and associated with type 2 diabetes and insulin resistance. This evidence concerns the gene CPT1B and type 2 diabetes mellitus.