IFNL4 and fibrosis: We show that in our cohort, patients who developed early post-transplant fibrosis had a 3.45 adjusted odds of having donor IFNL4 TT/TT genotype (p = 0.012, Table 3).These findings are consistent with the linked rs12979860 IFNL3 polymorphism (IL28B), where the favorable C/C genotype predicts increased post-transplant fibrosis when acquired in the donor liver [2,5].