Since ADPKD perturbs the Jnk/Stat (cJun N-terminal kinases/signal transducer and activator of transcription 1), MAPK (mitogen-activated protein kinase), mTOR (mammalian target of rapamycin), Wnt/Wgless (Wingless-related integration site/Wingless)and Erk (extracellular signal regulated kinase) kinase pathways [8], somatostatin analogues, vasopressin receptor antagonists, mTOR inhibitors, and statins have been tested for their ability to ameliorate the course of ADPKD. Here, SOAT1 is linked to autosomal dominant polycystic kidney disease.