FANCC and anemia (phenotype): This approach is exemplified by the observation that while Fancc‐/‐ mice do not develop bone marrow hypocellularity, the Fancc‐/‐ and Sod1‐/‐ double mutants develop this feature and go on to develop anemia and leucopenia, providing some evidence that oxidative stress contributes to bone marrow failure in FA 61.