Genome-wide, this analysis revealed that out of 456 genes prioritized for these two diseases, 136 had eQTLs, of which four genes (BLK, RASGRP1, SUOX, and GIN1) showed evidence for possible co-localization of GWAS signals and eQTLs in RA and two genes (BLK and SLC15A4) in SLE (see Figure S6 for examples). The gene discussed is SLC15A4; the disease is rheumatoid arthritis.