Investigation of IFNγ (100 ng/mL; 5.8 nM) in vitro has demonstrated ability to inhibit IL-4-induced CCL26 production from lung epithelial cells [12,15]; furthermore, IFNγ administered via the airways has demonstrated efficacy in mice at reducing airway eosinophilia [16]. The gene discussed is IFNG; the disease is Increased total eosinophil count.