Most importantly, we found that, in tissue samples from patients with thymoma-associated myasthenia gravis, the expression levels of miR-20b were correlated inversely with those of NFAT5 and CAMTA1, suggesting that the inhibitory regulation of NFAT5 and CAMTA1 by miR-20b could be implicated in the pathophysiology of thymoma and thymoma-associated myasthenia gravis. Here, NFAT5 is linked to myasthenia gravis.