HTT and Huntington disease: In the present study, as a first attempt to relate amygdala dysfunction and impulse control deficits in HD, we assessed in a within-subject design both risky decision-making and impulsivity as well as amygdala basal activity in early-symptomatic transgenic model of HD (tgHD) rats, a transgenic rodent model of HD carrying 51 CAG repeats under the control of the endogenous rat huntingtin promoter (Holzmann et al., 1998).