But we found that ectopically expressing miR-29c inhibitors without the treatment of TGF-β1 inhibited the mRNA expression of TTF-1 and E-cadherin, and enhanced the expression of vimentin and α-SMA, which indicated that the inhibition of miR-29c could be sufficient to induce EMT in lung cancer. This evidence concerns the gene ACTA1 and lung carcinoma.