Although both CD34+ HPCs and Kasumi-3 cells were utilized to establish latent HCMV infections in the present study and although the results showed a similar accumulation of miR-UL148D in host cells during late-stage HCMV infection, we must acknowledge that our mechanistic study of miR-UL148D’s role in HCMV latency was mainly performed in Kasumi-3 cells. This evidence concerns the gene CD34 and cytomegalovirus infection.