ACVR1B and disease arising from reactivation of latent virus: Moreover, while this manuscript was under review, Lau et al also reported that miR-UL148D is expressed during latency and demonstrated that miR-UL148D could limit proinflammatory cytokine secretion of latently infected cells by inhibiting ACVR1B expression[73], which suggests that miR-UL148D may exert multiple function to facilitate viral latent infection.