The A1/A2 polymorphism of the GpIIIa gene caused by a T-to-C nucleotide substitution at position 1565, which is associated with the occurrence of the amino acid Leu →Pro variant at residue 33 of the mature protein,17 has been widely studied in cardiovascular diseases.18 These studies have shown that possession of an A2 allele increases the risk for MI,19,20 CAD,21 and restenosis after stent placement.22 Eight patients had a GpIIIa gene polymorphism in our study. Here, ITGB3 is linked to cardiovascular disorder.