A potential role for HDAC9 in the pathogenesis of B-NHL is strengthened by its interaction with BCL6 (Petrie et al., 2003), a transcriptional repressor crucial for GC formation (Basso and Dalla-Favera, 2012) and whose deregulated expression in B cells leads to LPD and B-NHL (Cattoretti et al., 2005). The gene discussed is HDAC9; the disease is disseminated peritoneal leiomyomatosis.