The consumption of green tea has recently attracted much attention in the occidental culture because of its beneficial effects, such as protection of dopaminergic neurons from damage induced by 6-hydroxydopamine in a rat model of Parkinson's disease (Guo et al., 2007); reduction of mutant huntingtin misfolding and neurotoxicity in Huntington's disease models (Ehrnhoefer et al., 2006); direct protection of neurons against Aβ toxicity (Bastianetto et al., 2000); protection against Aβ-induced cognitive impairment in a rat model relevant to Alzheimer's disease (Haque et al., 2008). This evidence concerns the gene HTT and Huntington disease.