SLC7A2 and colitis: Taken together, these data show that protection from colitis in the C. rodentium model by Slc7a2 deletion is mediated by the recipient mouse genotype; this indicates that exacerbation of colitis due to SLC7A2 derives from the non-hematopoietic cells, consistent with the predominantly epithelial SLC7A2 localization that we have observed.